Tired of dark patches on the skin? Tired of using concealers all the time ?
Dr Jaishree will examine your skin and decide a treatment for your pigmentation, ranging from medicines to procedures. Procedures for pigmentation include Fractional Q switched Nd Yag laser and an array of peels such as Glycolic peels, TCA peel, Yellow peel, Obagi peel, Nomelan peel, Cosmelan peel and many more.
Clearlift laser :
Used exclusively with the Harmony XL, ClearLift represents a new alternative to traditional laser skin resurfacing and laser skin surgery
ClearLift improves fine lines, wrinkles, the appearance of small veins and redness caused by vascularity. It is gentle enough to treat areas that are typically too sensitive to treat with traditional ablative lasers, such as the delicate skin around the eyes, neck, mouth and chest.
Alma’s ClearLift delivers a controlled dermal wound without harming the overlying epidermis so that all stages of healing and skin repair occur under the intact epidermis.
Because ClearLift heats deep beneath the skin (up to 3mm in depth) no numbing is required prior to treatment and all skin healing happens without damaging the outer layer of skin.
Advantages: Fast and Easy to use – Treatments in as little as 20 minutes.
Virtually Painless, No Downtime
Visible improvement in skin appearance in as little as one treatment
Versatile – the 1064 nm Q-Switched module is also indicated for tattoo removal, pigmented and vascular lesions, nail fungus and more.
The Q-Switched laser module is indicated for use on all skin types (Fitzpatrick I – VI)
This potent stimulating peel confers longer lasting benefits beyond the temporary superficial improvements seen with ordinary peels. Peeling is very mild or even unnoticeable, with minimal to no downtime. Designed for acne, melasma, sun damage, fine lines, texture roughness, large pores, dullness. Promotes softer, smoother skin.
Suitable for any skin type or color
Usage studies show remarkable safety with minimal irritation
Can be repeated every 3 to 4 weeks if deeper repair is desired
What is the basic structure of the skin and what is responsible for giving it a color ?
Skin is the largest organ of the body. It is made up of two layers, the upper ‘epidermis’ and the lower ‘dermis’. The epidermis and the dermis are further divided into other layers. The lower most layer of the epidermis is known as the basal layer and it contains organelles called ‘melanosomes’. These melanosomes contain cells called melanocytes which produce a pigment called ‘melanin’. The color of skin depends mainly on this melanin and the amount of melanin present in the other layers of the epidermis. The thickness of epidermis and vascularity of the epidermis are other factors affecting the color of skin.
What are the causes of hypo-pigmentation (decrease in skin pigmentation)?
Pityriasis alba : Hypo pigmented patch may be seen on the face usually in young children. It is common in winter and in those with history of atopy. It can be treated with a mild topical steroid.
Pityriasis versicolor : It is a superficial fungal infection caused by a yeast ‘pityrosporum orbiculare’. Well defined hypo or hyper pigmented lesions with fine scales are found on the chest, back, neck and sometimes on the face. It is treated with topical and oral anti fungal drugs.
Leprosy : It is a chronic disease caused by ‘mycobacterium leprae’ and affects mainly the skin and nerves. A hypo pigmented patch with absent or decreased sensation is seen in tuberculoid type of leprosy (good immune response) whereas in lepromatous type (low immune response) the sensations may be normal. It can be cured with the help of drugs.
Vitiligo : This is an autoimmune diseases where de pigmented patches are seen, they may occur any where on the body. The treatment is detailed in the chapter on vitiligo.
secondary leucoderma : This occurs due to contact with certain chemicals phenol derivatives, glue of ‘bindi’, rubber slippers, plastic watch straps, photograph developing solutions or burns. It can be treated by spot dermabrasion or skin grafting.
Post inflammatory hypo pigmentation : It occurs after the healing phase of certain dermatoses such as eczemas, psoriasis, candidiasis etc.
Nevus depigmentosus : It is a congenital, well defined, hypo pigmented patch present since birth. It can be treated by spot dermabrasion or ablation with Co2 laser followed by ultra thin skin grafting.
What are the causes of hyper-pigmentation (increase in skin pigmentation)?
Hyper pigmentation may either be due to increased melanin deposition in the epidermis or dermis. Epidermal pigmentary disorders respond well to treatment while dermal pigmentation may take a long time to lighten.
Melasma : Seen as brown patches on the face, it is more commonly seen in females. It occurs due to hormonal changes in the body. The pigmentation increases on sun sun exposure.
Peri-orbital melanosis : Also known as ‘dark circles’ may be heriditary, due to stress or eye strain
Freckles and Lentigenes : These are tiny black spots on the face and are genetic in origin.
Photomelanosis : This is increased pigmentation due to sun exposure. The pigmentation occurs on exposed skin, commonly on the face, neck and the back. The pigmentation may be patchy or as diffused darkening of the exposed skin.
Sun burn (tan) : A condition commonly encountered in fair skinned people due to excessive sun exposure.
Post inflammatory hyper pigmentation : It may be seen in the following cases Seen after healing has occured, like in acne, eczemas, contact dermatitis etc. Drug induced pigmentation. Pigmentation due to cosmetics especially those containing fragrance.
Macular lichen amyloidosis.
Nevus or ‘Birth mark’ : Usually seen at birth but may also appear at a later age.
Tattoo : Tattoos are pigments inserted into the skin for decorative purpose which may at times be a part of tradition. Usually these artificial pigments are inserted deep into the dermis which makes them permanent and stubborn
What is the treatment for hyper-pigmented disorders?
Melasma, Photomelanosis and post inflammatory hyper pigmentation can be treated with topical creams such as hydroxy quinine, tretinoin, topical steroids, alpha hydroxy acid creams, azelic acid, arbutin, liquorice, kojic acid. If the patient does not improve with these medicines other modalities should be used, which may be :
Intense pulse light treatment
Laser treatment ( 532nm and 1064n Q switched Nd Yag laser )